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A scholar, mentor, colleague, and friend.
20th Annual Meeting Call for Proposals

We now seek proposals for symposia and keynote speakers for the 18th Annual Meeting of the SBN.

Welcome from the President

SBN President Cheryl Sisk.

I am delighted and honored to be your new President. SBN is a wonderful organization that provides significant benefits to its members. I look forward to helping the society meet your interests and needs in science and professional development, and I encourage you to share your ideas about how SBN can best serve you.

—Elizabeth Adkins-Regan

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The Society for Behavioral Neuroendocrinology offers four levels of eligibility for prospective members: Regular, Emeritus, Student, or Associate Memberships.

To see which membership class you qualify for, please review the membership eligibility requirements.

For additional information on SBN and the rules of membership, please see the SBN Bylaws.

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Elected Officers

PRESIDENT (2015-2017) Elizabeth Adkins-Regan

PRESIDENT-ELECT (2015-20175) Rae Silver

PAST PRESIDENT (2015-2017) Cheryl Sisk

SECRETARY (2015-2017) Colin John Saldanha

TREASURER (2013-2016) Nancy Forger

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Hormones and Behavior

Wednesday, October 07, 2015
Publication date: September 2015
Source:Hormones and Behavior, Volume 75

Author(s): Urs Kalbitzer, Michael Heistermann, Dorothy Cheney, Robert Seyfarth, Julia Fischer

In multi-male, multi-female groups of mammals, males usually compete aggressively over access to females. However, species vary in the intensity of male contest competition, which has been linked to differences in testosterone and glucocorticoid profiles. Chacma (Papio ursinus) and Guinea (P. papio) baboons constitute an intriguing model to examine variation in male competition and male endocrine correlates, because of the differences in their social systems. Chacma baboons live in stable female-bonded groups with linear male dominance hierarchies and a high male mating skew, whereas Guinea baboons live in male-bonded, multi-level societies. We recorded male behavior and assayed testosterone (fT) and glucocorticoid metabolite (fGC) levels from fecal samples in one population of each species. Male chacma baboons were more frequently involved in agonistic interactions, and dominance relationships were more consistent than in Guinea baboons, where we could not detect linear hierarchies. Notably, male chacma baboons were also more aggressive towards females, indicating an overall higher aggressiveness in this species. In contrast, male Guinea baboons showed higher levels of affiliative interactions and spatial tolerance. High-ranking and consorting male chacma baboons showed elevated fGC levels and also tended to show elevated fT levels, but there was no effect of consortship in Guinea baboons. Agonism was not related to hormone levels in either species. Thus, predictors of fT and fGC levels in Guinea baboons seem to differ from chacma baboons. Our results support the view that different social systems create differential selection pressures for male aggression, reflected by different hormone profiles.

Wednesday, October 07, 2015
Publication date: August 2015
Source:Hormones and Behavior, Volume 74

Author(s): Maya Frankfurt, Victoria Luine

This article is part of a Special Issue “Estradiol and Cognition”. Memory processing is presumed to depend on synaptic plasticity, which appears to have a role in mediating the acquisition, consolidation, and retention of memory. We have studied the relationship between estrogen, recognition memory, and dendritic spine density in the hippocampus and medial prefrontal cortex, areas critical for memory, across the lifespan in female rodents. The present paper reviews the literature on dendritic spine plasticity in mediating both short and long term memory, as well as the decreased memory that occurs with aging and Alzheimer's disease. It also addresses the role of acute and chronic estrogen treatments in these processes.

Wednesday, October 07, 2015
Publication date: August 2015
Source:Hormones and Behavior, Volume 74

Author(s): Carole Shum, Sara C. Macedo, Katherine Warre-Cornish, Graham Cocks, Jack Price, Deepak P. Srivastava

This article is part of a Special Issue “Estradiol and Cognition”. Over recent years tremendous progress has been made towards understanding the molecular and cellular mechanism by which estrogens exert enhancing effects on cognition, and how they act as a neuroprotective or neurotrophic agent in disease. Currently, much of this work has been carried out in animal models with only a limited number of studies using native human tissue or cells. Recent advances in stem cell technology now make it possible to reprogram somatic cells from humans into induced pluripotent stem cells (iPSCs), which can subsequently be differentiated into neurons of specific lineages. Importantly, the reprogramming of cells allows for the generation of iPSCs that retain the genetic “makeup” of the donor. Therefore, it is possible to generate iPSC-derived neurons from patients diagnosed with specific diseases, that harbor the complex genetic background associated with the disorder. Here, we review the iPSC technology and how it's currently being used to model neural development and neurological diseases. Furthermore, we explore whether this cellular system could be used to understand the role of estrogens in human neurons, and present preliminary data in support of this. We further suggest that the use of iPSC technology offers a novel system to not only further understand estrogens' effects in human cells, but also to investigate the mechanism by which estrogens are beneficial in disease. Developing a greater understanding of these mechanisms in native human cells will also aid in the development of safer and more effective estrogen-based therapeutics.

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